لایب سان

Ebook details:
عنوان: Cardiovascular Responses to Exercise: Second Edition (Colloquium Series on Integrated Systems Physiology From Mol)
نویسنده: Lusha Xiang, Robert L. Hester, D. Neil Granger
ناشر: Morgan & Claypool Life Sciences; 2 edition (December 1, 2016)
زبان: English
شابک: 1615047263, 978-1615047260
حجم: 15 Mb
فرمت: True Pdf

1. Capillary perfusion in skeletal muscle during exercise -- 1.1 Capillary perfusion at rest -- 1.1.1 Capillary vasomotion -- 1.1.2 Microcirculatory units -- 1.2 Regulation of capillary perfusion during exercise -- 1.2.1 Capillary perfusion during exercise -- 1.2.2 Factors determining capillary perfusion -- 1.3 Oxygen delivery to skeletal muscle during exercise -- 1.3.1 Oxygen gradient -- 1.3.2 Bohr effect -- 1.3.3 Oxygen deficient at onset of exercise. 2. Local control of arteriolar diameter and blood flow during exercise -- 2.1 Increased blood flow during exercise (functional hyperemia) -- 2.2 Blood flow control at the onset of exercise (phase I) -- 2.2.1 Oxygen delivery at the onset of exercise -- 2.2.2 Functional vasodilation and blood flow control at the onset of exercise -- 2.2.2.1 Muscle pump -- 2.2.2.2 Neurogenic vasodilation in skeletal muscle -- 2.2.3 Metabolic control (potassium) -- 2.2.4 Flow-mediated vasodilation -- 2.3 Blood flow control during steady-state dynamic exercise (phase II) -- 2.3.1 Metabolite control -- 2.3.1.1 Potassium -- 2.3.1.2 Oxygen -- 2.3.1.3 Hydrogen ion -- 2.3.1.4 Lactate -- 2.3.1.5 Adenosine -- 2.3.1.6 ATP -- 2.3.2 Endothelium-dependent vasoactive metabolites -- 2.3.2.1 Nitric oxide -- 2.3.2.2 Prostanoids -- 2.3.2.3 Endothelium-derived hyperpolarizing factors -- 2.3.3 Venular-arteriolar diffusion -- 2.3.3.1 Anatomy and "communication" of paired vessels -- 2.3.3.2 Regulation of arteriolar tone by paired venular endothelium-derived factors during exercise -- 2.3.3.3 Regulation of arteriolar tone by paired venular blood PO2 during exercise -- 2.3.3.4 Low venular PO2 and resultant increased ATP-induced production of prostanoids -- 2.3.4 Flow-mediated and conducted vasodilation -- 2.3.4.1 Flow-mediated vasodilation -- 2.3.4.2 Conducted vasodilation -- 2.3.5 Neural control of functional vasodilation -- 2.3.5.1 Motor nerve-mediated vasodilation -- 2.3.5.2 Vascular adrenoreceptors -- 2.3.5.3 Sympathetic escape. 3. Systemic control of cardiovascular response to exercise -- 3.1 Neural control mechanisms -- 3.1.1 Central command -- 3.1.2 Exercise pressor reflex -- 3.1.3 Baroreflex -- 3.2 Cardiac responses to exercise -- 3.2.1 Adrenergic and cholinergic receptors in heart -- 3.2.2 Heart rate -- 3.2.3 Stroke volume -- 3.2.4 Cardiac output -- 3.2.5 Cardiac muscle blood flow -- 3.3 Pulmonary hemodynamics in response to exercise -- 3.3.1. Pulmonary vessels, circulation, and gas exchange -- 3.3.1.1 Basic characteristics of pulmonary circulation -- 3.3.1.2 Pulmonary gas exchange -- 3.3.2. Pulmonary hemodynamic and oxygen kinetic responses during exercise -- 3.3.2.1 Pulmonary hemodynamic response to exercise -- 3.3.2.2 Lung oxygen uptake and blood gases during exercise -- 3.3.2.3 Right ventricle and exercise capability -- 3.4 Systemic hemodynamic responses to exercise -- 3.4.1 Brain blood flow during exercise -- 3.4.2 Renal blood flow during exercise -- 3.4.2.1 Regulation of renal blood flow and urine output under rest conditions -- 3.4.2.2 Regulation of renal blood flow and urine output during exercise. 4. Cardiovascular response to exercise under pathological conditions -- 4.1 Obesity and diabetics -- 4.1.1 Impaired local blood flow in the exercising muscle of obesity -- 4.1.1.1 Microvascular rarefaction in the exercising muscle of obesity -- 4.1.1.2 Impaired local control of functional vasodilation in the exercising muscle of obesity -- 4.1.1.3 Basal arteriolar tone and vascular remodeling in the exercising muscle of obesity -- 4.1.2 Impaired systemic control of cardiovascular response to exercise -- 4.1.2.1 Exercise presser reflex -- 4.1.2.2 Baroreflex resetting -- 4.1.2.3 Central command -- 4.1.2.4 Systemic hemodynamic responses -- 4.2 Other pathologic conditions -- 4.2.1 Coronary insufficiency and heart failure -- 4.2.1.1 Coronary insufficiency -- 4.2.1.2 Heart failure -- 4.2.2 McArdle's disease. References -- Author biography.

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